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Pathology and Pathogenesis The pathologic changes in VZV infection are unique and consist of one or more of the following: (1) an in ammatory reaction in several unilateral adjacent sensory ganglia of the spinal or cranial nerves, frequently of such intensity as to cause necrosis of all or part of the ganglion, with or without hemorrhage; (2) an in ammatory reaction in the spinal roots and peripheral nerve contiguous with the involved ganglia; (3) a less common poliomyelitis that closely resembles acute anterior poliomyelitis but is readily distinguished by its unilaterality, segmental localization, and greater involvement of the dorsal horn, root, and ganglion; and (4) a relatively mild leptomeningitis, largely limited to the involved spinal or cranial segments and nerve roots These pathologic changes are the substratum of the neuralgic pains, the pleocytosis, and the local palsies that may attend and follow the VZV infection.

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There may also be a delayed cerebral vasculitis (see further on) As to pathogenesis, herpes zoster represents a spontaneous reactivation of VZV infection, which becomes latent in the neurons of sensory ganglia following a primary infection with chickenpox (Hope-Simpson) This hypothesis is consistent with the differences in the clinical manifestations of chickenpox and herpes zoster, even though both are caused by the same virus Chickenpox is highly contagious by respiratory aerosol, has a well-marked seasonal incidence (winter and spring), and tends to occur in epidemics Zoster, on the other hand, is not communicable (except to a person who has not had chickenpox), occurs sporadically throughout the year, and shows no increase in incidence during epidemics of chickenpox In patients with zoster, there is practically always a past history of chickenpox.

Make sure you have good lighting as well as good technique to make sure that no solder bridges flow between the connections Finally locate and install U3, the LM386 IC audio amplifier integrated circuit Once again use of an IC socket is highly recommended Solder the IC socket to the PC board and insert the LM386 paying particular attention to the small notch or indented circle on the top of the IC body Pin 1 is to the left of the notch, cut-out or indented circle Take a short well-deserved break and when we return, we will install the remaining components on the PC board and finish up building the receiver Now locate power switch S1 Press S1 firmly into its six holes and solder all six pins The switch fits the board only one way Next, install J1, the RCA-style antenna jack and solder all four points.

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Such a history may be lacking in rare instances of herpes zoster in infants, but in these cases there has usually been prenatal maternal contact with VZV VZV DNA is localized primarily in trigeminal and thoracic ganglion cells, corresponding to the dermatomes in which chickenpox lesions are maximal and that are most commonly involved by VZV (Mahalingam et al) The supposition is that in both zoster and varicella infections the virus makes its way from the cutaneous.

Now, install J2, the subminiature headphone jack Solder all three points Be gentle and patient in inserting, so as not to damage the solder tabs Locate all three potentiometers and we will install them now Potentiometer controls R1, R2 and R3 are all mounted in a row next to switch S1 Insert the three PC mount potentiometers into their positions Check that the controls are pressed in firmly and straight against the top of the board Solder the three center pins and then the two larger mechanical mounting tabs Use enough solder for a solid connection The circuit board may require a few jumpers; cut component leads make good jumpers Insert the jumpers and solder into place, remember to trim the excess leads We are now down to the home-stretch.

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4. Slow Stochastics will be located above or below the chart. It should look something like Figure 3.5.

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