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which defines the deceleration parameter qo as (6.14) The larger the value of qo, the more rapid the deceleration. The simplest situation is if the Universe is matter dominated, p = O. Remember that by 'matter' we mean any pressureless material; it could be a collection of elementary particles, or equally well a collection of galaxies. Then from the acceleration equation (3.18) and the definition of critical density, equation (6.4), we find (6.15) So in this case, a measurement of qo would immediately tell us 0 0 . If we know the properties of the matter in the Universe, then qo is not independent of the first two parameters we have discussed, H o and 0 0 , Those two are sufficient to describe all the possibilities. However, we don't know everything about the material in the Universe, so qo can provide a new way of looking at the Universe. It can in principle be measured directly by making observations of objects at very large distances, such as the numbers of distant galaxies, because the deceleration governs how large the Universe would be at an earlier time. Recently, the first convincing measurements of qo have been made by two research groups studying distant supernovae of a class known as type la, which are believed to be good standard candles. To widespread surprise, the result is that the Universe appears to be accelerating at present, qo < 0. 1 None of the cosmological models that we have discussed so far are capable of satisfying this condition, as can be seen directly from the acceleration equation (3.18). This result is becoming finnly established, and is amongst the most dramatic observational results in modern cosmology. The following chapter discusses how to extend our simple cosmological models to account for it.

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1. Draw one or more paths around the portions of the image that you want to appear opaque. Areas outside the paths will be transparent. 2. Save the paths. Double-click the Work Path item in the Paths palette, enter a path name, and press Enter. (Try to use a name that will make sense three years from now when you have to revisit this document and determine what the heck you did.) 3. Choose the Clipping Path command from the Paths palette menu, as shown in Figure 8-40. Photoshop displays the dialog box shown at the top of the figure, asking you to select the saved paths you want to assign as the clipping path. Remember, you can t make the Work Path a clipping path; you must save it as a named path first.

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metabolic effects of leptin and adiponectin are summarized in Figure 1012 Adipose tissue contains numerous macrophages and other in ammatory cells (Weisberg et al, 2003; 4) When activated, for example by infections or hypoxia, these cells secrete numerous cytokines, notably TNF- and IL-6 (Pickup, 2004) These acute-phase in ammatory mediators have various effects, including insulin resistance and activation of the sympathetic nervous system, hypothalamopituitary-adrenal (HPA) axis and the reninangiotensin-aldosterone system (RAS) (Van Gaal, Mertens and De Block, 2006; Axelrod and Reisine, 1984; Chan, Tong and Critchley, 2002) This causes widespread pro-in ammatory, prothrombotic and haemodynamic changes as well as neuroendocrine disturbances and insulin resistance Consistent with this notion, chronic low-grade infections such as hepatitis B and C have been implicated in the development of type 2 diabetes (Pradhan et al, 2001; Mehta et al, 2000) and cardiac and renal complications (Lo et al.

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, 2004; Cheng et al, 2006; Soma et al, 2000), especially in obese subjects and in non-Caucasian populations (Lao et al, 2003; Ratziu, Trabut and Poynard, 2004) Two important pro-in ammatory cytokines are TNF- and IL-6 TNF- has long been implicated in in ammatory conditions such as rheumatoid arthritis and also in the catabolic states associated with various malignancies (cancer cachexia) and severe heart failure (Aggarwal, 2000) TNF- expression is increased in adipose tissue of obese humans and rodents (Hotamisligil, Shargill and Spiegelman, 1993; Hotamisligil et al, 1995) and can contribute to insulin resistance, either at a paracrine level on adjacent adipocytes, or at distant sites TNF- has been shown to inhibit the phosphorylation of speci c tyrosine residues on the insulin receptor and IRS-1 that normally follows insulin-receptor binding (Hotamisligil et al, 1994).

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IL-6 is secreted mainly by immune cells but also by adipose tissue, notably abdominal subcutaneous and visceral depots, which contribute up to one-third of the body s total IL-6 production (Mohamed-Ali et al, 1997) Circulating IL-6 concentrations correlate with fat mass and BMI, and also with insulin resistance (Bastard et al, 2000; Vozarova et al, 2001; Kern et al, 2001) In omental adipocytes, IL-6 (in the presence of dexamethasone) enhances leptin expression, while reducing expression of LPL; these effects,.

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