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The OS will load normally, using all drivers and Registry values. The OS will load normally and will log boot activities in a text file called BOOTLOG.TXT. The OS will load with minimal drivers (mouse, keyboard, and standard VGA). Same as Safe mode but with network drivers and configurations to allow network access. The OS will load normally but will allow you to selectively process or skip individual AUTOEXEC.BAT and CONFIG.SYS lines. Begins the boot process normally but stops before the protected-mode boot phase. Loads in MS-DOS mode without the real-mode configuration files. The only files loaded are IO.SYS, MSDOS.SYS, and COMMAND.COM. Loads an earlier version of DOS than the one that is included with Windows 9x. Can be used only if a previous version of DOS exists on the system.

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Wired network access provides a more or less reliable connection to the network. That is not the case in wireless networks, where the bearers might be inaccessible for shorter or longer periods of time due to fading, lost radio coverage, or deficient capacity. If you have ever lost a connection when you were driving in your car, you will know just how frustrating this can be. The architects of the WAP protocols infrastructure, when putting together the specifications for WAP, have taken the problem of connection stability into account and have designed into the layers the following features: M The sessions supported by the Session layer are assumed to be long-lived, so the problem of lost connections is addressed by allowing lost sessions to be resumed, even when dynamically assigned IP addresses are used. The Wireless Transaction Protocol (WTP) layer in WAP has been kept very simple compared to TCP, which is used on the wired Internet. Since no connection is set up, the effects of lost connections and other times of inactivity are minimized. The unpredictable nature of a wireless connection means that small segments of a message are often lost, and WTP supports selective retransmission of data, meaning that only the lost segments are retransmitted and not the entire message as in TCP.

Violent trauma to the spinal canal or skull can cause arachnoidal and dural tears. The associated neural injury dominates the picture and the dural tear may require repair in order to minimize the development of meningitis. More dif cult to understand is the occurrence of spinal cord herniation through an apparently spontaneous rent in the adjacent dura with no preceding injury. In view of the fact that we have encountered ve such instances in a decade, without trauma having occurred, it is probably not rare. In the typical case, a vertically oriented tear of limited extent occurs in the ventral dura, in the mid- or high-thoracic region, and a segment of the spinal cord protrudes through it into the epidural space. The result is a painless, subacute, and incomplete spinal cord syndrome, which reaches a plateau and leaves the patient with an asymmetrical spastic paraparesis and variable sensory loss. There are reports of a Brown-Sequard hemicord syndrome. Orthostatic headache of low-CSF pressure is not usually part of the syndrome. MRI or CT myelopathy demonstrates the protruded segment of the cord where it buckles through the dura. Presumably the herniation creates a suf cient degree of local ischemia or mechanical disturbance to account for the myelopathic symptoms. Surgical replacement of the cord in its proper position and repair of the tear has resulted in partial return of neurologic function (Vallee et al). As to the cause, a congenital duplication of the dura membranes combined with herniation through the inner layer has been observed in some cases at operation.

This form of craniocerebral trauma in infants is well known in large emergency practices but can be missed if not speci cally considered. As the name implies, the inciting trauma is typically violent shaking of the body or head of an infant, resulting in rapid acceleration and deceleration of the cranium combined with cervical whiplash. The presence of this type of injury must often be inferred from the combination of lesions on imaging studies or autopsy examination, but precision in examination is paramount because of its forensic and legal implications. The diagnosis is suspected from the combination of subdural hematomas and retinal hemorrhages, as summarized by Bonnier and colleagues. Sometimes there are occult skull fractures, but more often, there is little or no direct cranial trauma. Additional lesions may be evident on diffusion-

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