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The goals of performance and portability remain in conflict with security architecture goals. We reach the surprising conclusion that some goals are, despite all of the vendor promises in the world, fundamentally opposed to secure design. The best course for an application architect lies in acknowledging this fact and acting to mitigate it, rather than ignoring the conflicts or compromising quality by sacrificing security to other architectural goals. Recognition of the conflicts at an early stage affords applications and their owners the opportunity of doing things differently perhaps buying more hardware, changing database definitions, reorganizing user groups, reworking the network architecture, or switching vendor products. Conflicts left unresolved at least have the virtue of documentation, along with the possibility that at some future time the evolution of the application and the products it depends upon will result in resolution of the conflict. In the following sections, we will expand upon each of these goals and its relationship to security to explain why we cannot achieve perfection.



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The dimensions offered by the Window Size pop-up menu describe the entire editable area of a page. The Document window has been carefully designed to match specifications set by the primary browsers. Both the left and right margins are the same width as both the Netscape and Microsoft browsers, and the status bar matches the height of the browser s bottom row as well. The height of any given browser environment depends on which toolbars are being used; however, Dreamweaver s menu bar is the same height as the browsers menu bars.





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throw invalid_argument(s.str()); } // Does not record "do nothing" action. if(!contracts) return 0.0; // Determines type of transaction (buy or sell). int sgn = contracts > 0 1 : -1; if(!isOpen() || longClosedShort() == sgn) { // Either establishes new or increases existing position. cp_.push_back(PriceCostContractsIndex(price, cost, contracts, index)); return 0.0; } // Needs to offset at least partly existing position. double eqBeg = openEquity(price, pricePointValue); int n = contracts; while(cp_.size() != 0) { if(abs(cp_[0].contracts_) == abs(n)) { ts.push_back(Trade(cp_[0].price_, cp_[0].cost_, cp_[0].contracts_, cp_[0].index_, price, cost, index, pricePointValue)); cp_.pop_front(); return eqBeg - openEquity(price, pricePointValue); } else if(abs(cp_[0].contracts_) > abs(n)) { ts.push_back(Trade(cp_[0].price_, cp_[0].cost_, -n, cp_[0].index_, price, cost, index, pricePointValue)); cp_[0].contracts_ += n; return eqBeg - openEquity(price, pricePointValue); } else { ts.push_back(Trade(cp_[0].price_, cp_[0].cost_, cp_[0].contracts_, cp_[0].index_, price, cost, index, pricePointValue)); n += cp_[0].contracts_; cp_.pop_front(); } } // while(cp_.size() != 0) cp_.push_back(PriceCostContractsIndex(price, cost, n, index)); return eqBeg;

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in cholesterol synthesis) have shown that they not only lead to large and sustained reductions in both total and LDL-cholesterol in plasma but also to signi cant reductions in both heart disease and total mortality in both primary (Shepherd et al 1995) and secondary (SSSS Group 1994) intervention trials both in subjects with and without previous episodes of diagnosed heart disease The general assumption is that the decline in blood cholesterol caused by statins leads to the reductions in mortality and thus that cholesterol-lowering by other means should produce the same bene ts It is now widely accepted that lowering plasma cholesterol is universally bene cial There were, however, several very large studies published in the early 1990s which suggested that very low plasma cholesterol concentrations were associated with increased total mortality (due to increased non-cardiovascular causes (eg Jacobs et al 1992; Neaton et al 1992).

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In editorial comment upon these ndings, Hulley et al (1992) questioned whether universal promotion of cholesterol-lowering could still be ethically justi ed, especially in women Phytosterols not only reduce the absorption of cholesterol and its concentration in blood, they also reduce the blood levels of some other lipid soluble substances, in particular the carotenoids and vitamin E The effect of this reduction in absorption of these antioxidant molecules (see 5) is unknown There are a few people with rare genetic defects who are susceptible to high phytosterol intakes (such as those with phytosterolaemia described earlier in this section) The impact of stanols on these people is unknown and it is not established whether the larger number of people who are heterozygous for such conditions may also have some increased susceptibility to large phytosterol intakes.

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