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2 2 2 -(zr - H e ) -(zr + H e ) Q -y exp + exp exp 2s y2 us zs y 2p 2s x2 2s z2



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Regardless of whether the behavioral constellation of ADHD is the result of genetics or other causes, several neuropsychological constructs have been implicated as playing roles in the development and manifestation of ADHD. Some of these constructs with empirical support include sustained attention, behavioral inhibition, working memory, and planning ability; these abilities often are included within the multifaceted construct called executive function (Barkley, 1997; Stefanatos & Baron, 2007). It follows that speci c areas and structures of the brain that may be involved in ADHD include the frontal lobe, mesocortical pathway from the substantia nigra to the prefrontal cortex, mesolimbic pathway, nigrostiatal pathway, basal ganglia, limbic system, thalamus, corpus callosum, and cerebellum (Krain & Castellanos, 2006; Nigg, 2005). Broadly speaking, evidence suggests that ADHD is related to frontal lobe dysfunction and abnormal connections between the frontal lobe and subcortical areas of the brain (Biederman, 2005). See Figure 6.1 for an illustration of some of the more well-documented neurological correlates and pathways contributing to ADHD. Swanson et al. (2007) provided a review of the literature supporting the dopamine de cit hypothesis, which postulates that dysfunction of the neurotransmitter dopamine is a causal factor in the development of ADHD. The authors explain that dopamine is involved in carrying out some of the behaviors affected by ADHD, such as planning and self-regulation. In addition, the authors review the literature on other dopamine-related biological factors contributing to ADHD, citing ndings such as: Compared to control participants, individuals with ADHD have smaller brain structures such as the caudate nucleus and globus pallidus. In addition, children with ADHD show reduced blood ow and metabolism in the frontal lobes; and stimulant medications, such as methylphenidate, reduce ADHD symptoms by increasing dopamine levels. The pattern of ndings





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// Release the lock when the guard goes out of scope, // but only if <acquire> succeeded. ~Guard () { if (owner_) lock_->release (); } private: // Pointer to the lock we're managing. LOCK *lock_;

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Extension to the ILTS: As for the bisimulation relation, the simulation relation can be extended to initialized labeled transition systems as follows: LT S, s0 simulates LT S , s0 if there is a simulation relation connecting their respective initial states: i.e. there is a simulation relation R containing (s0 , s0 ). Now, the co-simulation concept de nes a relation of equivalence using the simulation pre-order. DEFINITION 12.31 (Co-simulation). LT S co-simulates LT S Def LT S, simulates LT S and LT S simulates LT S.

[Odell 98] Odell, James. Advanced Object-Oriented Analysis and Design Using UML. New York: SIGS Books, 1998.

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Given that u i . / is concave, this can be solved by maximizing a Lagrangian ! k X X X i i LD u i .x / pj xj Cj

will write an incomplete contract. Then, when the state of nature is realized, they will renegotiate the contract, since at this stage they know what kind of good should be traded. (115)

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