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i believe jquery datatable comes with all you need check it out https://datatables. net/extensions/buttons/examples/initialisation/export.html. Renal hypercalciuria occurs when the renal tubules are unable to efficiently reabsorb filtered calcium, and hypercalciuria results Spilling calcium in the urine results in secondary hyperparathyroidism Serum calcium typically is normal Thiazides are effective long-term therapy in patients with this disorder 2 Hyperuricosuric Hyperuricosuric calcium nephrolithiasis is secondary to dietary excesses or uric acid metabolic defects Both disorders can be treated with purine dietary restrictions or allopurinol therapy (or both) In contrast to uric acid nephrolithiasis, patients with hyperuricosuric calcium stones typically maintain a urinary pH greater than 55 Monosodium urates absorb and adsorb inhibitors and promote heterogeneous nucleation Hyperuricosuric calcium nephrolithiasis is probably secondary to epitaxy, or heterogeneous nucleation In such situations, similar crystal structures (ie, uric acid and calcium oxalate) can grow together with the aid of a protein matrix infrastructure 3 Hyperoxaluric Hyperoxaluric calcium nephrolithiasis is usually due to primary intestinal disorders Patients usually present with a history of chronic diarrhea frequently associated with inflammatory bowel disease or steatorrhea Increased bowel fat combines with intraluminal calcium to form a soap-like product Calcium is therefore unavailable to bind to oxalate, which is then freely and rapidly absorbed A small increase in oxalate absorption will significantly increase stone formation If the diarrhea or steatorrhea cannot be effectively curtailed, oral calcium supplements should be given with meals More than 2 g/d of ascorbic acid will increase urinary oxalate levels Emphasis on encouraging increased fluid intake is required for these patients as for all stone-formers 4 Hypocitraturic Hypocitraturic calcium nephrolithiasis may be secondary to chronic diarrhea, type I (distal) renal tubular acidosis, chronic hydrochlorothiazide treatment, and, in rare cases, is idiopathic Any condition that results in metabolic acidosis (including prolonged fasting, hypomagnesemia, and hypokalemia) will decrease urinary citrate excretion, since it will be consumed by the citric acid cycle within the mitochondria of proximal tubular cells Hypocitraturia is frequently associated with other forms of calcium stone formation Citrate appears to bind to calcium in solution, thereby decreasing available calcium for stone formation Potassium citrate supplements are usually effective Urinary citrate is decreased in acidosis and is increased during alkalosis The potassium will supplement the frequently associated hypokalemic states, and citrate will help to correct the acidosis A typical dose is 20 mEq three times a day (available in solution and in 10-mEq waxed tablets), or 30 mEq of crystal formulations twice a day. convert pdf to excel using javascript: Export data from pdf form to excel (JavaScript) - Acrobat Answers convert pdf to excel using javascript PDF to Excel conversion , PDF/A-3, better support for JavaScript ...
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Sample Javascript code for using PDFTron SDK to convert Office documents to PDF (including Word, Excel , PowerPoint and Publisher) without needing any ... 1 Hypercalciuric Hypercalciuric calcium nephrolithiasis (> 200 mg/24 h; > 4 mg/kg/24 h) can be caused by absorptive, resorptive, and renal disorders Absorptive hypercalciuria is secondary to increased absorption of calcium at the level of the small bowel, predominantly in the jejunum, and can be further subdivided into types I, II, and III Type I absorptive hypercalciuria is independent of calcium intake There is increased urinary calcium on a regular or even a calcium-restricted diet Treatment is centered upon decreasing bowel absorption of calcium Cellulose phosphate, a chelating agent, is an effective form of therapy An average dose is 10 15 g in three divided doses It binds to the calcium and impedes small bowel absorption due to its increased bulk Cellulose phosphate does not change the intestinal transport mechanism It should be given with meals so it will be available to bind to the calcium Taking this chelating agent prior to bedtime is ineffective Postmenopausal women should be treated with caution It is interesting, however, that there is no enhanced decline in bone density after long-term use Inappropriate use without an initial metabolic evaluation (see above) may result in a negative calcium balance and a secondary parathyroid stimulation Long-term use without follow-up metabolic surveillance may result in hypomagnesuria and secondary hyperoxaluria and recurrent calculi Routine follow-up every 6 8 months will help encourage medical compliance and permit adjustments in medical therapy based upon repeat metabolic studies Thiazide therapy is an alternative to cellulose phosphate in the treatment of type I absorptive hypercalciuria Thiazides decrease renal calcium excretion but have no impact on intestinal absorption This therapy results in increased bone density of approximately 1% per year Thiazides have limited long-term utility (< 5 years) since they may lose their hypocalciuric effect with continued therapy Type II absorptive hypercalciuria is diet dependent Decreasing calcium intake by 50% (approximately 400 mg/ d) will decrease the hypercalciuria to normal values (150 200 mg/24 h) There is no specific medical therapy Type III absorptive hypercalciuria is secondary to a renal phosphate leak This results in increased vitamin D synthesis and secondarily increased small bowel absorption of calcium This can be readily reversed by orthophosphates (250 mg three to four times per day) Orthophosphates do not change intestinal absorption but rather inhibit vitamin D synthesis Resorptive hypercalciuria is secondary to hyperparathyroidism Hypercalcemia, hypophosphatemia, hypercalciuria, and an elevated PTH value are found Appropriate surgical resection of the parathyroid adenoma cures the disease and the urinary stones Medical management is invariably a failure. pdf to excel javascript Cloud API - PDF To Excel - JavaScript - Convert PDF To XLS From ...
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